Avalon Medical Clinic

We extend our deepest gratitude to Dr. Iqra Azad for creating this exceptionally insightful and well-researched comparison — a valuable resource that meaningfully benefits both the general public and the healthcare community.

Are you worried about hair loss??
The future of hair loss treatment is looking bright! For more than 30 years, treatment options for androgenetic alopecia (male and female pattern hair loss) have largely revolved around a small group of core medications: minoxidil and antiandrogens (finasteride/dutasteride, spironolactone).
​While these remain excellent treatments for many patients, the next generation of therapies is taking a completely different approach.
​So what's around the corner?
Researchers are studying a variety of new medications that work quite differently from our current standard treatments
​Some block androgen receptors directly rather than simply lowering DHT. Clascoterone is a topical androgen receptor antagonist that blocks DHT from activating the follicle. GT20029 is a compound that targets the androgen receptor for degradation, removing the "lock" that DHT normally binds to.
​Others target entirely new hormonal pathways. Medications such as HMI-115 are investigating whether blocking prolactin signaling can promote hair growth—a completely different approach from traditional anti-androgen therapies.
​Several therapies aim to wake up dormant hair follicles. Drugs like PP405 are designed to activate resting follicle stem cells by altering their metabolism. Rather than simply slowing hair loss, the goal is to encourage follicles to begin producing hair again.
​Regenerative injectable therapies, including AMP-303, are being developed to stimulate follicles to re-enter the active growth (anagen) phase.
​Researchers are also exploring therapies that target cellular metabolism, thyroid hormone signaling, potassium channels, and even biological aging pathways using medications such as rapamycin and metformin.
​It's important to remember that many of these therapies are still in clinical trials and some may never make it to the real world for use by patients. Some may make it to market but will need to prove themselves against current therapies. I believe we're entering one of the most exciting eras in hair loss research!
​Reference of Interest
Agrawal A et al. Emerging pharmacotherapies for androgenetic alopecia. Expert Opin Pharmacother. 2026.

Warmest congratulations to you on Canada Day, may it be filled with joy and pride 🎉🇨🇦🎊

Healthy skin starts with the right ingredients — not guesswork.
At Avalon Medical Clinic, we guide you toward evidence‑based skincare tailored to your unique concerns.
From brightening to hydration to acne care, these essentials help you build a routine that truly works.

AS YOU KNOW 👇

While LED light therapy (Low-Level Light Therapy or LLLT) is a legitimate modality in clinical practice, not everything in this chart is true or scientifically proven.This graphic is a classic example of consumer marketing, blending some established science with significant exaggerations and pseudoscientific claims.
Here is a breakdown of the claims versus the clinical reality, colour-coded to match the chart:
🟢 The Evidence-Based (With Caveats)
🔴 Red Light (630nm - 660nm):There is robust clinical evidence supporting red light for stimulating fibroblast activity, enhancing collagen production, and reducing inflammation. It is effective for mild anti-aging and wound healing. However, claims like "whiten pale spot" are inaccurate.
🔵 Blue Light (415nm - 470nm): This also has solid clinical backing. Blue light creates reactive oxygen species that destroy Cutibacterium acnes, making it an effective adjunctive treatment for mild to moderate inflammatory acne. However, calling it an "acne eraser" that repairs "without leaving any scars" is an overstatement of its preventative capabilities.
🟡 The Questionable and Exaggerated
🟡 Yellow Light (590nm): While sometimes used clinically to reduce erythema (redness) and improve circulation, the claim that it "Increases Immunity" or "decompose[s] pigment" is not supported by standard dermatological literature.
🟢 Green Light (520nm): Often marketed for targeting melanocytes and reducing hyperpigmentation, the clinical data for green LED light is very weak compared to topical treatments or actual clinical lasers. Claims about balancing "water and oil" are purely marketing speak.
🟣 Purple Light: This is simply a combination of red and blue LEDs. While it theoretically offers the benefits of both, it will not "heal acne scars" (atrophic scars), which typically require resurfacing techniques like microneedling or fractional lasers to remodel tissue.
🩵 Cyan Light: There is little to no credible clinical evidence supporting cyan light for "increasing cell energy" or "face whitening."
🔴 The Misleading
⚪ Laser Light": This is a major misnomer. Consumer masks use LEDs (Light Emitting Diodes), which produce non-coherent light. True lasers produce coherent, focused light and have entirely different tissue interactions, safety profiles, and clinical endpoints (as you would see with a clinical Nd:YAG or Alexandrite laser). An LED mask cannot "decompose stain and improve sagging skin" to the degree implied.
📸 The Before & After Photos:The images provided in the chart appear heavily manipulated, relying on different lighting, angles, and makeup to show drastic changes. Consumer-grade LED masks operate at a fraction of the energy output of clinical devices, and the results are generally subtle and require consistent, long-term use.
In short, while red and blue light have legitimate applications in skin health, the rest of the chart overpromises the capabilities of a home-use LED device.